Accumulating mitochondrial DNA mutations drive premature hematopoietic aging phenotypes distinct from physiological stem cell aging.
Gudmundur Norddahl, Kees-Jan Pronk, Martin Wahlestedt, Gerd Sten, Jens Nygren, Amol Ugale, Mikael Sigvardsson, David Bryder
Research output: Contribution to journal › Article › peer-review