Abstract
The nonobese diabetic mouse is highly susceptible not only to diabetes but to several autoimmune diseases, and one might suspect that these are controlled by a shared set of genes. However, based on various gene-segregation experiments, it seems that only a few loci are shared and that each disorder is influenced also by a unique set of genes.
Original language | English |
---|---|
Pages (from-to) | 203-213 |
Journal | Scandinavian Journal of Immunology |
Volume | 57 |
Issue number | 3 |
DOIs | |
Publication status | Published - 2003 |
Subject classification (UKÄ)
- Immunology in the medical area