The Relationship Between Cardiac Reactivity in the Laboratory and in Real Life

Derek Johnston; Martti Tuomisto; Geoffrey Patching

doi:10.1037/0278-6133.27.1.34

The Relationship Between Cardiac Reactivity in the Laboratory and in Real Life

Derek Johnston, Martti Tuomisto, Geoffrey Patching

Stockholm University

Research output: Contribution to journal › Article › peer-review

Abstract

Objective: An excessive cardiovascular response to acute stress is a probable risk factor for cardiovascular (CV) disease. Such reactivity is usually assessed from the CV response to laboratory stressors.
However, if it is a risk factor, correlated responses must occur in real life. Design: In the present study, we investigated the relationship between the heart rate (HR) response to five laboratory stressors and HR reactivity in the field. Measures: HR variation, the response to a real life stressor (public speaking), and
the increase in HR with periods of self-reported tense arousal. Ambulatory HR, activity and posture were measured continuously over a 7-hr period. Results: The HR increase to laboratory stressors did not relate to HR variation consistently, but it did relate to the other two field measures. Conclusion: The results suggested that a tendency to increased HR reactivity may be a risk factor for cardiovascular disease when combined with exposure to stress.

Original language	English
Pages (from-to)	34-42
Journal	Health Psychology
Volume	27
Issue number	1
DOIs	https://doi.org/10.1037/0278-6133.27.1.34
Publication status	Published - 2008
Externally published	Yes

Subject classification (UKÄ)

Psychology

Free keywords

real life stressors
laboratory stressors
heart rate
cardiac reactivity
public speaking

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

Access to Document

10.1037/0278-6133.27.1.34

Cite this

@article{d86ab420d7d9413784da07804405e512,

title = "The Relationship Between Cardiac Reactivity in the Laboratory and in Real Life",

abstract = "Objective: An excessive cardiovascular response to acute stress is a probable risk factor for cardiovascular (CV) disease. Such reactivity is usually assessed from the CV response to laboratory stressors. However, if it is a risk factor, correlated responses must occur in real life. Design: In the present study, we investigated the relationship between the heart rate (HR) response to five laboratory stressors and HR reactivity in the field. Measures: HR variation, the response to a real life stressor (public speaking), and the increase in HR with periods of self-reported tense arousal. Ambulatory HR, activity and posture were measured continuously over a 7-hr period. Results: The HR increase to laboratory stressors did not relate to HR variation consistently, but it did relate to the other two field measures. Conclusion: The results suggested that a tendency to increased HR reactivity may be a risk factor for cardiovascular disease when combined with exposure to stress.",

keywords = "real life stressors, laboratory stressors, heart rate, cardiac reactivity, public speaking",

author = "Derek Johnston and Martti Tuomisto and Geoffrey Patching",

year = "2008",

doi = "10.1037/0278-6133.27.1.34",

language = "English",

volume = "27",

pages = "34--42",

journal = "Health Psychology",

issn = "0278-6133",

publisher = "American Psychological Association (APA)",

number = "1",

}

TY - JOUR

T1 - The Relationship Between Cardiac Reactivity in the Laboratory and in Real Life

AU - Johnston, Derek

AU - Tuomisto, Martti

AU - Patching, Geoffrey

PY - 2008

Y1 - 2008

N2 - Objective: An excessive cardiovascular response to acute stress is a probable risk factor for cardiovascular (CV) disease. Such reactivity is usually assessed from the CV response to laboratory stressors. However, if it is a risk factor, correlated responses must occur in real life. Design: In the present study, we investigated the relationship between the heart rate (HR) response to five laboratory stressors and HR reactivity in the field. Measures: HR variation, the response to a real life stressor (public speaking), and the increase in HR with periods of self-reported tense arousal. Ambulatory HR, activity and posture were measured continuously over a 7-hr period. Results: The HR increase to laboratory stressors did not relate to HR variation consistently, but it did relate to the other two field measures. Conclusion: The results suggested that a tendency to increased HR reactivity may be a risk factor for cardiovascular disease when combined with exposure to stress.

AB - Objective: An excessive cardiovascular response to acute stress is a probable risk factor for cardiovascular (CV) disease. Such reactivity is usually assessed from the CV response to laboratory stressors. However, if it is a risk factor, correlated responses must occur in real life. Design: In the present study, we investigated the relationship between the heart rate (HR) response to five laboratory stressors and HR reactivity in the field. Measures: HR variation, the response to a real life stressor (public speaking), and the increase in HR with periods of self-reported tense arousal. Ambulatory HR, activity and posture were measured continuously over a 7-hr period. Results: The HR increase to laboratory stressors did not relate to HR variation consistently, but it did relate to the other two field measures. Conclusion: The results suggested that a tendency to increased HR reactivity may be a risk factor for cardiovascular disease when combined with exposure to stress.

KW - real life stressors

KW - laboratory stressors

KW - heart rate

KW - cardiac reactivity

KW - public speaking

U2 - 10.1037/0278-6133.27.1.34

DO - 10.1037/0278-6133.27.1.34

M3 - Article

SN - 0278-6133

VL - 27

SP - 34

EP - 42

JO - Health Psychology

JF - Health Psychology

IS - 1

ER -

The Relationship Between Cardiac Reactivity in the Laboratory and in Real Life

Abstract

Subject classification (UKÄ)

Free keywords

UN SDGs

Access to Document

Fingerprint

Cite this