Martin Wahlestedt

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  1. 2017
  2. The slippery slope of hematopoietic stem cell aging

    Martin Wahlestedt & David Bryder, 2017 Dec 1, In : Experimental Hematology. 56, p. 1-6 6 p.

    Research output: Contribution to journalArticle

  3. Critical Modulation of Hematopoietic Lineage Fate by Hepatic Leukemia Factor

    Martin Wahlestedt, Ladopoulos, V., Isabel Hidalgo, Sanchez Castillo, M., Hannah, R., Petter Säwén, Haixia Wan, Monika Dudenhöffer-Pfeifer, Mattias Magnusson, Norddahl, G. L., Göttgens, B. & David Bryder, 2017 Nov 21, In : Cell Reports. 21, 8, p. 2251-2263 13 p.

    Research output: Contribution to journalArticle

  4. Clonal reversal of ageing-associated stem cell lineage bias via a pluripotent intermediate

    Martin Wahlestedt, Eva Erlandsson, Trine Kristiansen, Lu, R., Brakebusch, C., Weissman, I. L., Joan Yuan, Martin-Gonzalez, J. & David Bryder, 2017 Feb 22, In : Nature Communications. 8, 14533.

    Research output: Contribution to journalArticle

  5. Induction of the 5S RNP–Mdm2–p53 ribosomal stress pathway delays the initiation but fails to eradicate established murine acute myeloid leukemia

    Jaako, P., A. Ugale, M. Wahlestedt, Velasco, T., Cammenga, J., Lindström, M. S. & D. Bryder, 2017 Jan, In : Leukemia. 31, 1, p. 213-221

    Research output: Contribution to journalArticle

  6. MLL-ENL-mediated leukemia initiation at the interface of lymphoid commitment

    A. Ugale, P. Säwén, M. Dudenhöffer-Pfeifer, M. Wahlestedt, Norddahl, G. L. & D. Bryder, 2017, In : Oncogene. 36, p. 3207-3212

    Research output: Contribution to journalArticle

  7. 2015
  8. Concise Review: Hematopoietic Stem Cell Aging and the Prospects for Rejuvenation.

    Martin Wahlestedt, Pronk, C. J. & David Bryder, 2015, In : Stem cells translational medicine. 4, 2, p. 186-194

    Research output: Contribution to journalArticle

  9. 2014
  10. Hematopoiesis: Functional Insights by Cell Fate Conversion

    Martin Wahlestedt, 2014, Stem Cell Aging. 164 p.

    Research output: ThesisDoctoral Thesis (compilation)

  11. Hematopoietic Stem Cells Are Intrinsically Protected against MLL-ENL-Mediated Transformation.

    Amol Ugale, Norddahl, G., Martin Wahlestedt, Petter Säwén, Jaako, P., Kees-Jan Pronk, Shamit Soneji, Cammenga, J. & David Bryder, 2014, In : Cell Reports. 9, 4, p. 1246-1255

    Research output: Contribution to journalArticle

  12. Induced Hematopoietic Stem Cells: Unlocking Restrictions in Lineage Potential and Self-renewal.

    Martin Wahlestedt & David Bryder, 2014, In : Cell Stem Cell. 14, 5, p. 555-556

    Research output: Contribution to journalDebate/Note/Editorial

  13. Somatic cells with a heavy mitochondrial DNA mutational load render iPS cells with distinct differentiation defects.

    Martin Wahlestedt, Ameur, A., Moraghebi, R., Norddahl, G., Sten, G., Niels-Bjarne Woods & David Bryder, 2014, In : Stem Cells. 32, 5, p. 1173-1182

    Research output: Contribution to journalArticle

  14. 2013
  15. An epigenetic component of hematopoietic stem cell aging amenable to reprogramming into a young state.

    Martin Wahlestedt, Norddahl, G., Sten, G., Amol Ugale, Micha Frisk, M-A., Mattsson, R., Tomas Deierborg, Mikael Sigvardsson & David Bryder, 2013, In : Blood. 121, 21, p. 4257-4264

    Research output: Contribution to journalArticle

  16. SOCS2 is dispensable for BCR/ABL1-induced chronic myeloid leukemia-like disease and for normal hematopoietic stem cell function.

    Hansen, N., Helena Ågerstam, Martin Wahlestedt, Niklas Landberg, Askmyr, M., Mats Ehinger, Rissler, M., Henrik Lilljebjörn, Johnels, P., Ishiko, J., Melo, J. V., Whalen, A., David Bryder, Marcus Järås & Thoas Fioretos, 2013, In : Leukemia. 27, p. 130-135

    Research output: Contribution to journalArticle

  17. 2012
  18. 2011