Clinical measurements of proprioception, muscle strength and laxity in relation to function in the ACL-injured knee.

Research output: Contribution to journalArticle


A knee injury with anterior cruciate ligament (ACL) rupture may cause deficits in proprioception, increased laxity and decreased muscle strength. Although it may be common knowledge that these factors affect knee function, only a few studies have been performed where this has been investigated in the clinical situation, and the results are not conclusive. The purpose of this study was therefore to investigate how and to what extent proprioception, laxity and strength affect knee joint function and evaluate if the methods commonly used for estimating these factors clinically seem to be relevant. The study encompassed 36 patients with ACL deficiency. A single-leg hop test for distance and subjective rating of knee function were defined as dependent variables and analyzed separately in stepwise linear regression models where proprioception, knee joint laxity, hamstrings and quadriceps strength, age and sex were defined as independent variables. Higher threshold values (poorer proprioception), increased side-to-side difference of anterior laxity and poorer strength significantly predicted shorter length of the hop test. Higher rating of subjective function corresponded to female gender, lesser side-to-side difference of anterior laxity and better proprioception.


Research areas and keywords

Subject classification (UKÄ) – MANDATORY

  • Surgery


  • Laxity, Proprioception, Knee joint, Anterior cruciate ligament, Knee injuries
Original languageEnglish
Pages (from-to)41533
JournalKnee Surgery, Sports Traumatology, Arthroscopy
Issue number1
Publication statusPublished - 2007
Publication categoryResearch

Bibliographic note

The information about affiliations in this record was updated in December 2015. The record was previously connected to the following departments: Department of Orthopaedics (Lund) (013028000), Division of Physiotherapy (Closed 2012) (013042000), Clinical Neurophysiology (013013001)

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