Endogenous IFN-β signaling exerts anti-inflammatory actions in experimentally induced focal cerebral ischemia.
Research output: Contribution to journal › Article
Interferon (IFN)-β exerts anti-inflammatory effects, coupled to remarkable neurological improvements in multiple sclerosis, a neuroinflammatory condition of the central nervous system. Analogously, it has been hypothesized that IFN-β, by limiting inflammation, decreases neuronal death and promotes functional recovery after stroke. However, the core actions of endogenous IFN-β signaling in stroke are unclear.
|Research areas and keywords||
Subject classification (UKÄ) – MANDATORY
|Journal||Journal of Neuroinflammation|
|Publication status||Published - 2015|
The information about affiliations in this record was updated in December 2015. The record was previously connected to the following departments: Neuroinflammation (013210006), Faculty of Medicine (000022000), Laboratory for Experimental Brain Research (013041000)