Human endogenous peptide p33 inhibits detrimental effects of LL-37 on osteoblast viability.
Research output: Contribution to journal › Article
High levels of the antimicrobial peptide, LL-37, are detected in gingival crevicular fluid from patients with chronic periodontitis. LL-37 not only shows antimicrobial activity but also affects host-cell viability. The objective of the present study was to identify endogenous mechanisms that antagonize the detrimental effects of LL-37 on osteoblast viability, focusing on the human peptide p33 expressed on the surface of various cell types.
|Research areas and keywords||
Subject classification (UKÄ) – MANDATORY
|Journal||Journal of Periodontal Research|
|Publication status||Published - 2015|
The information about affiliations in this record was updated in December 2015. The record was previously connected to the following departments: Vascular Physiology (013212034), Mucosal biology (013212033), Division of Infection Medicine (BMC) (013024020)
Related research output
Daniel Svensson, 2017, Lund: Lund University: Faculty of Medicine. 166 p.
Research output: Thesis › Doctoral Thesis (compilation)
2013/11/01 → …