Induction of T helper 2 responses against human Apolipoprotein B100 does not affect atherosclerosis in ApoE-/- mice.
Research output: Contribution to journal › Article
Immune responses against LDL antigens have been found to play an important modulatory role in atherosclerosis. Immunization with homologous oxidized LDL, as well as human apolipoprotein B100 (ApoB)-derived peptides, inhibit atherosclerosis in hypercholesterolemic animal models of atherosclerosis. However, the role of antigen-specific Th2 responses in atherosclerosis remains to be fully clarified.
|Research areas and keywords||
Subject classification (UKÄ) – MANDATORY
|Early online date||2014 May 27|
|Publication status||Published - 2014|
Related research output
Sara Rattik, 2015, Experimental Cardiovascular Research Unit, Lund University. 183 p.
Research output: Thesis › Doctoral Thesis (compilation)