Induction of T helper 2 responses against human Apolipoprotein B100 does not affect atherosclerosis in ApoE-/- mice.

Research output: Contribution to journalArticle

Abstract

Immune responses against LDL antigens have been found to play an important modulatory role in atherosclerosis. Immunization with homologous oxidized LDL, as well as human apolipoprotein B100 (ApoB)-derived peptides, inhibit atherosclerosis in hypercholesterolemic animal models of atherosclerosis. However, the role of antigen-specific Th2 responses in atherosclerosis remains to be fully clarified.

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Subject classification (UKÄ) – MANDATORY

  • Cardiac and Cardiovascular Systems
Original languageEnglish
JournalCardiovascular Research
Early online date2014 May 27
Publication statusPublished - 2014
Publication categoryResearch
Peer-reviewedYes

Related research output

Sara Rattik, 2015, Experimental Cardiovascular Research Unit, Lund University. 183 p.

Research output: ThesisDoctoral Thesis (compilation)

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