Induction of T helper 2 responses against human Apolipoprotein B100 does not affect atherosclerosis in ApoE-/- mice.

Forskningsoutput: TidskriftsbidragArtikel i vetenskaplig tidskrift

Abstract

Immune responses against LDL antigens have been found to play an important modulatory role in atherosclerosis. Immunization with homologous oxidized LDL, as well as human apolipoprotein B100 (ApoB)-derived peptides, inhibit atherosclerosis in hypercholesterolemic animal models of atherosclerosis. However, the role of antigen-specific Th2 responses in atherosclerosis remains to be fully clarified.

Detaljer

Författare
Enheter & grupper
Forskningsområden

Ämnesklassifikation (UKÄ) – OBLIGATORISK

  • Kardiologi
Originalspråkengelska
TidskriftCardiovascular Research
Tidigt onlinedatum2014 maj 27
StatusPublished - 2014
PublikationskategoriForskning
Peer review utfördJa

Relaterad forskningsoutput

Sara Rattik, 2015, Experimental Cardiovascular Research Unit, Lund University. 183 s.

Forskningsoutput: AvhandlingDoktorsavhandling (sammanläggning)

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