Kristina Rydell-Törmänen

Telefon+46462229441
E-postadresskristina.rydell-tormanenmed.luse

Om du gjort några ändringar i Pure kommer de visas här snart.

Nyckelord

Experimentell lungmedicin
Lungfibros, IPF
Kroniskt obstruktiv lungsjukdom, KOL
Regenerativ medicin

Fingeravtryck

Utforska forskningsämnen där Kristina Rydell-Törmänen är aktiv. Dessa ämnesetiketter kommer från personens arbeten. Tillsammans bildar de ett unikt fingeravtryck.

1 Liknande profiler

21 Artikel i vetenskaplig tidskrift
2 Översiktsartikel
1 Kapitel samlingsverk
1 Debate/Note/Editorial
Mer
- 1 Doktorsavhandling (sammanläggning)

Preclinical evidence for the role of stem/stromal cells in COPD
Bölükbas, D., Augusto Silva, I., Rydell-Törmänen, K. & Wagner, D., 2019, Stem Cell-Based Therapy for Lung Disease. Burgess, J. K. & Heijink, I. H. (red.). 1 uppl. Springer International Publishing, s. 73-96 24 s.
Forskningsoutput: Kapitel i bok/rapport/Conference proceeding › Kapitel samlingsverk › Forskning › Peer review
The Applicability of Mouse Models to the Study of Human Disease
Rydell-Törmänen, K. & Johnson, J. R., 2019, I: Methods in molecular biology (Clifton, N.J.). 1940, s. 3-22 20 s.
Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review
Effects of 5-Hydroxytryptamine Class 2 Receptor Antagonists on Bronchoconstriction and Pulmonary Remodeling Processes
Löfdahl, A., Wenglén, C., Rydell-Törmänen, K., Westergren-Thorsson, G. & Larsson-Callerfelt, A. K., 2018 maj 1, I: American Journal of Pathology. 188, 5, s. 1113-1119 7 s.
Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review
Pulmonary fibrosis in vivo displays increased p21 expression reduced by 5-HT_2B receptor antagonists in vitro - A potential pathway affecting proliferation
Löfdahl, A., Rydell-Törmänen, K., Larsson-Callerfelt, A. K., Wenglén, C. & Westergren-Thorsson, G., 2018 dec. 1, I: Scientific Reports. 8, 1, 1927.
Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review

Öppen tillgång
Uncontrolled asthmatics have increased FceRI⁺ and TGF-β–positive MC_TC mast cells and collagen VI in the alveolar parenchyma
Andersson, C. K., Weitoft, M., Rydell-Törmänen, K., Bjermer, L., Westergren-Thorsson, G. & Erjefält, J. S., 2018 mars 1, I: Clinical and Experimental Allergy. 48, 3, s. 266-277 12 s.
Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review

The cellular interaction with matrix in chronic pulmonary diseases
Westergren-Thorsson, G., Löfdahl, A., Larsson Callerfelt, A. & Rydell-Törmänen, K.
2016/11/01 → …
Projekt: Avhandling

Kristina Rydell-Törmänen

Personlig profil

Nyckelord

Fingeravtryck

Nätverk

Se detaljer

Preclinical evidence for the role of stem/stromal cells in COPD

The Applicability of Mouse Models to the Study of Human Disease

Effects of 5-Hydroxytryptamine Class 2 Receptor Antagonists on Bronchoconstriction and Pulmonary Remodeling Processes

Pulmonary fibrosis in vivo displays increased p21 expression reduced by 5-HT_2B receptor antagonists in vitro - A potential pathway affecting proliferation

Uncontrolled asthmatics have increased FceRI⁺ and TGF-β–positive MC_TC mast cells and collagen VI in the alveolar parenchyma

The cellular interaction with matrix in chronic pulmonary diseases

Kristina Rydell-Törmänen

Personlig profil

Nyckelord

Fingeravtryck

Nätverk

Forskningsoutput

Preclinical evidence for the role of stem/stromal cells in COPD

The Applicability of Mouse Models to the Study of Human Disease

Effects of 5-Hydroxytryptamine Class 2 Receptor Antagonists on Bronchoconstriction and Pulmonary Remodeling Processes

Pulmonary fibrosis in vivo displays increased p21 expression reduced by 5-HT2B receptor antagonists in vitro - A potential pathway affecting proliferation

Uncontrolled asthmatics have increased FceRI+ and TGF-β–positive MCTC mast cells and collagen VI in the alveolar parenchyma

Projekt

The cellular interaction with matrix in chronic pulmonary diseases

Pulmonary fibrosis in vivo displays increased p21 expression reduced by 5-HT_2B receptor antagonists in vitro - A potential pathway affecting proliferation

Uncontrolled asthmatics have increased FceRI⁺ and TGF-β–positive MC_TC mast cells and collagen VI in the alveolar parenchyma