Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Ben King; Klaudia Kulak; Ulrika Krus; Rebecca Rosberg; Ewelina Golec; katarzyna wozniak; Maria Gomez; Enming Zhang; David J O'Connell; Erik Renström; Anna Blom

doi:10.1016/j.cmet.2018.09.009

Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Ben King, Klaudia Kulak, Ulrika Krus, Rebecca Rosberg, Ewelina Golec, katarzyna wozniak, Maria Gomez, Enming Zhang, David J O'Connell, Erik Renström, Anna Blom

Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review

49 Citeringar (SciVal)

Originalspråk	engelska
Sidor (från-till)	202-210.e6
Tidskrift	Cell Metabolism
Volym	29
Utgåva	1
DOI	https://doi.org/10.1016/j.cmet.2018.09.009
Status	Published - 2019 jan. 8

Ämnesklassifikation (UKÄ)

Cell- och molekylärbiologi
Endokrinologi och diabetes

Tillgång till dokument

10.1016/j.cmet.2018.09.009

49 Citeringar
1 Doktorsavhandling (sammanläggning)

Innate immunity in diabetes mellitus. Complement components C4BP and C3 promote survival of β cells under metabolic challenges.
Kulak, K., 2021, Lund: Lund University, Faculty of Medicine. 69 s.
Forskningsoutput: Avhandling › Doktorsavhandling (sammanläggning)

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King, Ben ; Kulak, Klaudia ; Krus, Ulrika ; Rosberg, Rebecca ; Golec, Ewelina ; wozniak, katarzyna ; Gomez, Maria ; Zhang, Enming ; O'Connell, David J ; Renström, Erik ; Blom, Anna. / Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation. I: Cell Metabolism. 2019 ; Vol. 29, Nr. 1. s. 202-210.e6.

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title = "Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation",

author = "Ben King and Klaudia Kulak and Ulrika Krus and Rebecca Rosberg and Ewelina Golec and katarzyna wozniak and Maria Gomez and Enming Zhang and O'Connell, {David J} and Erik Renstr{\"o}m and Anna Blom",

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doi = "10.1016/j.cmet.2018.09.009",

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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation. / King, Ben; Kulak, Klaudia; Krus, Ulrika; Rosberg, Rebecca ; Golec, Ewelina; wozniak, katarzyna; Gomez, Maria ; Zhang, Enming; O'Connell, David J; Renström, Erik ; Blom, Anna.

I: Cell Metabolism, Vol. 29, Nr. 1, 08.01.2019, s. 202-210.e6.

Forskningsoutput: Tidskriftsbidrag › Artikel i vetenskaplig tidskrift › Peer review

TY - JOUR

T1 - Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

AU - King, Ben

AU - Kulak, Klaudia

AU - Krus, Ulrika

AU - Rosberg, Rebecca

AU - Golec, Ewelina

AU - wozniak, katarzyna

AU - Gomez, Maria

AU - Zhang, Enming

AU - O'Connell, David J

AU - Renström, Erik

AU - Blom, Anna

PY - 2019/1/8

Y1 - 2019/1/8

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DO - 10.1016/j.cmet.2018.09.009

M3 - Article

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VL - 29

SP - 202-210.e6

JO - Cell Metabolism

JF - Cell Metabolism

SN - 1550-4131

IS - 1

ER -

Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Ämnesklassifikation (UKÄ)

Tillgång till dokument

Forskningsoutput

Innate immunity in diabetes mellitus. Complement components C4BP and C3 promote survival of β cells under metabolic challenges.

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