Sammanfattning
The nonobese diabetic mouse is highly susceptible not only to diabetes but to several autoimmune diseases, and one might suspect that these are controlled by a shared set of genes. However, based on various gene-segregation experiments, it seems that only a few loci are shared and that each disorder is influenced also by a unique set of genes.
Originalspråk | engelska |
---|---|
Sidor (från-till) | 203-213 |
Tidskrift | Scandinavian Journal of Immunology |
Volym | 57 |
Nummer | 3 |
DOI | |
Status | Published - 2003 |
Ämnesklassifikation (UKÄ)
- Immunologi inom det medicinska området