Human endogenous peptide p33 inhibits detrimental effects of LL-37 on osteoblast viability.

Daniel Svensson, Johannes Westman, Claes Wickström, Daniel Jönsson, Heiko Herwald, Bengt-Olof Nilsson

Forskningsoutput: TidskriftsbidragArtikel i vetenskaplig tidskriftPeer review

9 Citeringar (SciVal)

Sammanfattning

High levels of the antimicrobial peptide, LL-37, are detected in gingival crevicular fluid from patients with chronic periodontitis. LL-37 not only shows antimicrobial activity but also affects host-cell viability. The objective of the present study was to identify endogenous mechanisms that antagonize the detrimental effects of LL-37 on osteoblast viability, focusing on the human peptide p33 expressed on the surface of various cell types.
Originalspråkengelska
Sidor (från-till)80-88
TidskriftJournal of Periodontal Research
Volym50
Utgåva1
DOI
StatusPublished - 2015

Bibliografisk information

The information about affiliations in this record was updated in December 2015.
The record was previously connected to the following departments: Vascular Physiology (013212034), Mucosal biology (013212033), Division of Infection Medicine (BMC) (013024020)

Ämnesklassifikation (UKÄ)

  • Odontologi

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