Mucosal glucagon-like peptide-1 and gastric inhibitory polypeptide cell numbers in the super-obese human foregut after gastric bypass.

Bent J Nergård, Andreas Lindqvist, Hjörtur G Gislason, Leif Groop, Mikael Ekelund, Nils Wierup, Jan Hedenbro

Forskningsoutput: TidskriftsbidragArtikel i vetenskaplig tidskriftPeer review

Sammanfattning

Super-obesity, a body mass index>50 kg/m(2), is difficult to treat. Many studies have focused on the anatomic changes of the intestines; the physiologic background is not clearly identified. It is established that Roux-en-Y gastric bypass (RYGB) augments secretion of glucagon-like peptide-1 (GLP-1), peptide tyrosine tyrosine (PYY), and insulin, but other aspects of gut hormone cell function in the alimentary limb are unknown.
Originalspråkengelska
Sidor (från-till)1237-1246
TidskriftSurgery for Obesity and Related Diseases
Volym11
Nummer6
DOI
StatusPublished - 2015

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